Selected publication list

1.     Resident tissue macrophages: Key coordinators of tissue homeostasis beyond immunity. Zhao, J., Andreev, I., Silva, H.M. 2024. Science Immunology. 10.1126/sciimmunol.add1967

2.     c-MAF dependent perivascular macrophages regulate diet induced metabolic syndrome. Silva, H.M., Kitoko, J.Z., Queiroz, C.P., Kroehling, L., Matheis, F., Yang, K.L., Ren-Fielding, C., Littman, D.R., Bozza, M.T., Mucida, D., and Lafaille, J.J. 2021. Science Immunology. 10.1126/sciimmunol.abg7506

3.     Leukocyte Heterogeneity in Adipose Tissue, Including in Obesity. Weinstock, A, Moura Silva, H, Moore, KJ, Schmidt, AM, Fisher, EA. 2020. Circ Res 126, 1590-1612. doi: 10.1161/CIRCRESAHA.120.316203. PMID:32437300

4.     Vasculature-associated fat macrophages readily adapt to inflammatory and metabolic challenges. Silva, HM, Báfica, A, Rodrigues-Luiz, GF, Chi, J, Santos, PDA, Reis, BS, Hoytema van Konijnenburg, DP, Crane, A, Arifa, RDN, Martin, P et al.. 2019. J Exp Med 216, 786-806. doi:10.1084/jem.20181049. PMID:30862706

5.     CX3CR1+ monocytes modulate learning and learning-dependent dendritic spine remodeling via TNF-α. Garré, JM, Silva, HM, Lafaille, JJ, Yang, G. 2017. Nat Med 23, 714-722. doi: 10.1038/nm.4340. PMID:28504723

6.     P2X7 receptor inhibition ameliorates dendritic spine pathology and social behavioral deficits in Rett syndrome mice. Garré, JM, Silva, HM, Lafaille, JJ, Yang, G. 2020. Nat Commun 11, 1784.  doi: 10.1038/s41467-020-15590-5. PMID:32286307

7.     Niche-Selective Inhibition of Pathogenic Th17 Cells by Targeting Metabolic Redundancy. Wu, L, Hollinshead, KER, Hao, Y, Au, C, Kroehling, L, Ng, C, Lin, WY, Li, D, Silva, HM, Shin, J et al.. 2020. Cell 182, 641-654.e20. doi: 10.1016/j.cell.2020.06.014. PMID:32615085

8. Serum Amyloid A Proteins Induce Pathogenic Th17 Cells and Promote Inflammatory Disease. Lee, JY, Hall, JA, Kroehling, L, Wu, L, Najar, T, Nguyen, HH, Lin, WY, Yeung, ST, Silva, HM, Li, D et al.. 2020. Cell 180, 79-91.e16. doi: 10.1016/j.cell.2019.11.026 PMID:31866067

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https://www.ncbi.nlm.nih.gov/myncbi/1jW0dalgm9jk-/bibliography/public/